By R. Seth
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Extra info for Absinthe-minded: Then and now
Tests. , 2003), fertility as well as the display of intromissive and ejaculatory behaviors was reduced, though not eliminated, in ArKO male mice. , 2001). Interestingly, this deficit in fertility was reversed in ArKO males that received neonatal injections of E, suggesting that the observed deficits in male sexual behavior and resultant fertility resulted from the lack of the perinatal, organizational actions of estrogens formed via neural aromatization as opposed to a deficiency in the activational actions of estrogens on the adult expression of sexual behavior.
1989. Lesions of the SDN‐POA inhibit sexual behavior of male Wistar rats. Brain Res Bull 23: 483-492. De Vries GJ, Simerly RB. 2002. Anatomy, development, and function of sexually dimorphic neural circuits in the mammalian brain. Hormones, Brain and Behavior. Pfaff DW, Arnold AP, Etgen AM, Fahrbach SE, Rubin RT, editors. San Diego: Academic Press; pp. 137-192. De Vries GJ, Rissman EF, Simerly RB, Yang LY, Scordalakes EM, et al. 2002. A model system for study of sex chromosome effects on sexually dimorphic neural and behavioral traits.
Effects of testosterone propionate implants in the brain on the sexual behavior and peripheral tissue of the male rat. Physiol Behav 11: 633-640. Kindon HA, Baum MJ, Paredes RJ. 1996. Medial preoptic/ anterior hypothalamic lesions induce a female‐typical profile of sexual partner preference in male ferrets. Horm Behav 30: 514-527. Krege JH, Hodgin JB, Couse JF, Enmark E, Warner M, et al. 1998. Generation and reproductive phenotypes of mice lacking estrogen receptor beta. Proc Natl Acad Sci USA 95: 15677-15682.
Absinthe-minded: Then and now by R. Seth